NeuroCalm FLX+

Neural optics system

00LightCinematic opening

Light is more than what we see.

Every photon begins a biological journey: through the retina, along neural pathways and into the networks involved in how we experience light.

01RetinaInside the retina

The eye detects. The brain interprets.

Three systems. Interconnected responses.

Scroll sequence · select a type to explore

Rods
Vision under low-light conditions.
Cones
Colour and detailed visual perception.
ipRGCs
Melanopsin-expressing retinal neurons involved in non-image-forming light responses, with connections to several brain pathways.

Automatic sequence: rods, cones, then ipRGCs.

Educational schematic of retinal cell types. Proportions are not to scale.

02PathwaysFrom retina to brain

Light does not stop at the retina.

Image-forming visual pathway

01Rods and cones signal through bipolar cells to retinal ganglion cells, the output neurons of the eye.

Read the full pathway

Image-forming visual pathway

Rods and cones feed retinal ganglion cells whose axons form the optic nerve. Nasal fibres cross at the optic chiasm, so each optic tract carries the opposite half of the visual field to the lateral geniculate nucleus, and the optic radiation delivers it to the primary visual cortex along the calcarine sulcus. This is the route that builds the image you see.

Caution Educational schematic: proportions and positions are approximate. The exact proportion of crossing fibres and the size of V1 vary considerably between individuals.

03ExperienceTypical processing versus photophobia

The same light. A different experience.

Photophobia is more than light intensity. Visual, sensory and pain-related neural pathways may interact.

Typical light processing

  • Rods and cones drive vision through the lateral geniculate nucleus to V1.
  • Melanopsin ganglion cells (ipRGCs), which also receive rod and cone input, signal brightness to the suprachiasmatic nucleus and the pretectum.
  • Signal amplitude here is illustrative only; nothing represents a measurement of any brain.
Mechanism notes

Typical light processing

Calm, low-amplitude illustrative signalling along established routes.

Light reaches the brain through more than one route. Rods and cones build the image through the lateral geniculate nucleus and primary visual cortex, while melanopsin ganglion cells signal overall brightness to the circadian clock and the pupil reflex. In this mode the illustration keeps every signal calm and low in amplitude, with clear pathway labels.

Conceptual interaction diagramRetina, with melanopsin ganglion cells receiving rod and cone input, sends a proposed (dashed) projection to the posterior thalamus. Trigeminal input from the dura arrives at the same thalamic region by the established trigeminovascular route, and a proposed ocular-surface route is dashed. The posterior thalamus projects to somatosensory, insular and visual cortex. In photophobia-associated mode the trigeminal and thalamic nodes are emphasised. Conceptual diagram, not a measurement.RetinaipRGC + rod / cone inputDuratrigeminovascularOcular surfacetrigeminal, ophthalmic branchPosterior thalamusconvergence (rat Po/LP; human pulvinar)CortexS1 / insula / visualestablishedproposed
Conceptual diagram, not a measurement.

Overcast daylightconceptual

Soft, diffuse daylight, conceptually. Cones take over vision and melanopsin cells begin to report overall brightness. Emphasis moves toward the image-forming and circadian routes.

Conceptual illustration only. Not a clinical sensitivity test, a physiological model or a representation of any patient's neural response. Screen brightness is held nearly constant by design.

04AtlasThe neurology of light sensitivity

One symptom. Multiple possible mechanisms.

Some light sensitivity arises mainly in neural processing, some at the ocular surface or inside the eye, and some is mixed. The atlas marks each.

Select a condition

Choose one of the seven conditions to see where its light sensitivity is thought to arise, which structures light up in the schematic, and what is known versus still under investigation.

Atlas cautions (3)

Not a diagnostic tool. Light sensitivity is a symptom shared by many conditions, several of them urgent, and in one clinic series a quarter of patients left without a documented cause. The atlas explains proposed mechanisms; it does not identify anyone's condition.

Visual snow is distinct from migraine aura: different time course, imaging signature and treatment response.

Photosensitive epilepsy is not photophobia: it is light- or pattern-triggered seizures via occipital hyperexcitability, whereas photophobia is light-evoked discomfort or pain. The two are never merged here.

05SpectrumThe optical engineering opportunity

What if we could engineer the spectrum itself?

Scene: light from a dissolving brain re-forms as a 380 to 700 nanometre spectrum band with wavelength marks at 380, 450, 500, 550, 600, 650, 700 nanometres. A smoke grey explorer lens slides in front of it. With the lens inserted the whole spectrum dims to roughly a quarter of its brightness, the 460 to 490 and 585 to 600 nanometre regions dim further, 520 to 560 nanometres keeps a small relative peak, and the red end stays brightest.

Design-target bands

Values recited in the granted patent claims and specification; not independent measurement.

Band shows the transmitted result through the illustrative curve

The lens acts on light only, not on neurons or on the mechanisms of any condition.

460 to 490 nm520 to 560 nm585 to 600 nm0255075100380400450500550600650700Wavelength (nm)Spectral transmittance (%)

Spectral transmittance chart. Illustrative curve shaped to the band-averaged values in granted Indian Patent 587746 (FIG. 3, Table 2); not a spectrophotometer trace. Wavelength 380 to 700 nanometres on the horizontal axis, transmittance 0 to 100 percent on the vertical axis. 460 to 490 nm attenuated, 83 ± 2 percent (granted claim 1), mean 17 percent; 520 to 560 nm green comfort band, 23 to 25 percent transmission peak (granted claim 1), mean 24 percent; 585 to 600 nm attenuated, 78 ± 2 percent (granted claim 1), mean 22 percent. Use the left and right arrow keys to move the wavelength cursor by one nanometre, Shift with an arrow for ten, Home and End for the ends of the range, Enter or Space to select or deselect the band under the cursor, Escape to clear.

Illustrative curve shaped to the band-averaged values in granted Indian Patent 587746 (FIG. 3, Table 2); not a spectrophotometer traceAttenuated band (granted claim 1)Green band kept (granted claim 1)
Mean spectral transmittance per band. Illustrative curve shaped to the band-averaged values in granted Indian Patent 587746 (FIG. 3, Table 2); not a spectrophotometer trace.
BandRoleMean transmittance (percent)
460 to 490 nmAttenuated, 83 ± 2 percent (granted claim 1)17
520 to 560 nmGreen comfort band, 23 to 25 percent transmission peak (granted claim 1)24
585 to 600 nmAttenuated, 78 ± 2 percent (granted claim 1)22

Smoke grey tint, overall visible light transmission about 21 to 25 percent (patentee's figures; EN ISO 12312-1 Filter Category 2). Not suitable for driving in twilight or at night.

How to read this chart

Reading the chart: a clear lens would sit near 90 percent across the visible range. NeuroCalm FLX+ is a smoke grey tint, so its curve sits near 21 to 25 percent through most of the visible range, dips to about 17 percent at 460 to 490 nm and about 22 percent at 585 to 600 nm, holds a small peak of 23 to 25 percent at 520 to 560 nm, and rises only in the red. The depth of a dip shows how much is removed; its width shows selectivity.

Biology for context, not a lens property

Melanopsin, the photopigment of intrinsically photosensitive retinal ganglion cells, is most sensitive near 480 nm in vitro (human melanopsin peak about 479 nm). Measured at the cornea, the CIE S 026 melanopic action spectrum peaks near 490 nm, because the eye's own media filter short wavelengths before light reaches the retina.

The curve is an illustration shaped to the band-averaged values the granted specification reports (FIG. 3 bins, Table 2 spot values, claims 1 and 3): about 1 percent below 400 nm, 17 percent at 460 to 490 nm, a 23 to 25 percent peak at 520 to 560 nm, 22 percent at 585 to 600 nm, rising to about 47 percent in the red. The shape between those points is interpolated. It is not a spectrophotometer trace of NeuroCalm FLX+, and no percentage read from it is an independent measurement.

06NeuroCalm FLX+An Indian-granted optical invention

Introducing

NeuroCalm FLX+™

Patented dual-band wavelength-selective optical engineering.

Indian patent no.
587746
Application no.
202521094370
Inventor
Surajkumar Arunkumar Dubey (Suraj Dubey)
Patentee
Sleepaxa Private Limited
R&D
Acieon Labs
Status
Granted 27 April 2026, 10 claims

“An optical lens with dual-band selective light attenuation”

Verify on the Indian Patent Office register(manual search)

Patent filings and grants describe intellectual property protection. They do not by themselves represent clinical approval, medical-device clearance, diagnosis, treatment, or cure claims.

  • 460 to 490 nm, 83 percent attenuated
  • 520 to 560 nm, 23 to 25 percent peak kept
  • 585 to 600 nm, 78 percent attenuated

07AccessIndian-granted patented dual-band optical technology

Built on optical science. Experienced in person.

  1. I

    The invention

    Granted Indian patent and technical documentation.

    View the patent chapter
  2. II

    The science

    Spectral physics, human visual pathways and the published research on light sensitivity.

    Revisit the science
  3. III

    The experience

    Qualified offline optical partners for product trials and fitting.

    Find a partner

For professionals

Request technical documentation or express interest in a dispensing partnership.

connect@sleepaxa.com

Find a NeuroCalm experience partner

Discover verified participating locations as the network is published.

Participating locations will be published here as the partner network is verified. No addresses are listed until each location is confirmed.

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NeuroCalm FLX+™

An optical innovation by Acieon Labs / Sleepaxa Private Limited.

Educational content, not medical advice. A granted patent is not a certification of clinical efficacy. .